The Department of Psychiatry hosts a Grand Rounds lecture on Fridays, 12:00pm - 1:00pm (unless otherwise noted) in the WisPIC Commons room at WisPIC. In order to obtain CME credit, you must sign in at each conference. Annual CME statements are distributed every August.
Friday, July 26, 2013: Art Walaszek MD, Associate Professor and Vice Chair for Education. "What you need to know about ABPN Maintenance of Certification"
Friday, August 2, 2013: Heather Huang, MD, Clinical Asst. Prof., University of Wisconsin, Department of Psychiatry. “Integrated Care Models: Behavioral Health & Primary Care.”
Friday, August 9, 2013: Puru Thapa MD,MPH, Departments of Psychiatry and Epidemiology Section Chief, The Psychiatric Research Institute at the University of Arkansas for Medical Sciences. “Antipsychotics and Sudden Cardiac Death.”
Friday, August 23, 2013: Roger Kathol, MD, President, Cartesian Solutions, Inc. "Integrated Medical and Psychiatric Services: Adding Clinical and Economic Value in the Medical Setting."
Friday, September 6, 2013: Robert Golden, MD, Dean, School of Medicine and Public Health, University of Wisconsin. “The "Indolent" History of Serotonin and Depression.”
Friday, October 25, 2013: Mark Snowden MD, MPH, Associate Professor, Department of Psychiatry and Behavioral Sciences, University of Washington. “TBA”
Early exposure to severe stress in childhood increases the incidence of mood and anxiety disorders in adulthood. Far less researched, but of equal importance, is the theory that moderate early stress exposure instead of increasing vulnerability results in subsequent resilience. Variously described as inoculating, immunizing, steeling, or toughening, the notion that moderate postnatal stress exposure strengthens resistance to subsequent stressors has far-reaching implications for understanding the pathogenesis and prevention of stress-related affective disorders. Although the biological underpinnings of stress inoculation-induced resilience in humans are largely unknown, new insights have emerged from animal models. Evidence from these studies indicates that early exposure to moderate stressors that temporarily stimulate anxiety and activate the hypothalamic-pituitary-adrenal (HPA) axis lead subsequently to diminished negative arousal, prosocial tendencies, enhanced cognitive control, increased curiosity, larger prefrontal cortical volumes, higher cerebrospinal fluid oxytocin levels, and attenuated HPA axis activation. In contrast to rodents, rearing differences in the development of neuroendocrine stress resistance in monkeys are more closely related to differences in prior stress exposure than to differences in maternal care. Additionally, unlike in rodents, no rearing differences in glucocorticoid feedback sensitivity are observed in squirrel monkeys, suggesting that the neural basis of stress resistance in primates may differ from that in rodents. This assertion is supported by new evidence showing that benzodiazepine treatment prior to moderate stress exposure blocks the subsequent development of behavioral indications of resilience in a squirrel monkey model. Findings from these animal models support the intriguing hypothesis that moderate early stress exposure may likewise provide a pathway to stress resilience in human populations.
Friday, February 7, 2014: Jerry L. Halverson, MD, FAPA, Medical Director of Adult Services, Rogers Memorial Hospital. "WHIO measures."
Friday, March 7, 2014, 11:00am: Ryan Herringa, MD, Ph.D. Assistant Professor, Department of Psychiatry, University of Wisconsin. “Exploring neural mechanisms of pediatric post-traumatic stress disorder.”